COMPARING GOAL SETTING APPROACHES TO BOOST COMPUTER-RELATED PRO-ENVIRONMENTAL BEHAVIORS

This research focuses on improving individuals’ pro-environmental behaviors related to information technology and system use. To do so, we draw on and extend goal-setting theory by comparing three goal interventions: goal-setting, goal-setting plus implementation plans, and goal-setting with both implementation plans and visualization of success. Two longitudinal studies examine individuals’ selfset goals: the first examines employees’ computer-based electricity usage in the workplace over six weeks and the second utilizes a diary approach method over four weeks to examine the effects of different goal setting conditions on students’ environmental outcomes. Both studies find that setting goals increases pro-environmental behaviors. However, rebound effects can occur when interventions are removed. Visualization of success appears to reduce this rebound effect and we suggest that future research continue to investigate methods for reducing rebound, including the roles of values and multiple goals on the efficacy of goal-setting. This paper contributes to Green IS research in several ways: conceptually (by responding to calls for more theory-based research), methodologically (by measuring objective computer-based energy usage in study 1 and by utilizing a diary method in study 2), and practically (by demonstrating the effectiveness of visualization to goal setting)

Emodin Improves Intestinal Health and Immunity through Modulation of Gut Microbiota in Mice Infected by Pathogenic Escherichia coli O1

The effect of emodin on the intestinal mucosal barrier of a mouse E. coli O1-induced diarrhea model was observed. Following successful establishment of a diarrhea model, the mice were treated with drugs for seven days. Intestinal lesions and the shape and the number of goblet cells were assessed via hematoxylin-eosin and periodic-acid-Schiff staining, while changes in inflammatory factors, ultrastructure of the small intestine, expression of MUC-2, and changes in the intestinal microbiota were analyzed via RT-PCR, electron microscopy, immunofluorescence, and 16S rRNA sequencing. Examination showed that emodin ameliorated pathological damage to the intestines of diarrheic mice. RT-PCR indicated that emodin reduced TNF-α, IL-β, IL-6, MPO, and COX-2 mRNA levels in duodenal tissues and increased the levels of sIgA and MUC-2 and the number of goblet cells. Microbiome analysis revealed that Escherichia coli O1 reduced bacterial richness and altered the distribution pattern of bacterial communities at the phylum and order levels in cecum contents. Notably, pathogenic Clostridiales and Enterobacteriales were significantly increased in diarrheic mice. However, emodin reversed the trend. Thus, emodin protected against intestinal damage induced by E. coli O1 and improved intestinal mucosal barrier function in mice by increasing the abundance of beneficial intestinal microbiota and inhibiting the abundance of harmful bacteria, thereby alleviating diarrhea